AP39 and AP123 promoted Nrf2-regulated redox signaling pathway

Highlights • Mitochondrial targeted H2S donor AP123 restores endothelial function through a mechanism involving PI3KCREBeNOS axis • H2S availability is

AP123 treatment rescued eNOS expression, as well as NO levels, and restored p-CREB expression in both the HG environment and the presence of PAG

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